Ipreflavonoids yog hluavtaws flavonoids muab los ntawm daidzein. Ipreflavonoids zoo ib yam li cov kua isoflavones tiv thaiv pob txha, tab sis tsis muaj cov tshuaj estrogen zoo li isoflavones thiab yog li ntawd muaj kev phiv tsawg. Niaj hnub no, ipriflavone yog muag raws li kev noj zaub mov hauv khw muag khoom hauv ntau tshaj ob lub teb chaws suav nrog Tebchaws Meskas. Nyob rau hauv ltalis, Nyiv, thiab lwm yam, nws yog sivOsteoporosis: Kev tshawb fawb tau pom tias ipflavonoids tuaj yeem tiv thaiv thiab thim rov qab osteoporosis los ntawm kev txhawb nqa calcium nkag rau hauv cov pob txha thiab inhibiting pob txha tawg. Tsis tas li ntawd, nws ua rau cov pob txha ceev, txo qhov mob, thiab txo qhov kev pheej hmoo ntawm pob txha.
Hemiplegic Stroke thiab pob txha poob: Cov kev tshawb fawb soj ntsuam tau pom tias nyob rau hauv cov neeg mob uas tsis muaj vitamin D-tsis muaj hemiplegic stroke cov neeg mob, ipriflavone ua ke nrog vitamin D tiv thaiv cov pob txha tsis zoo dua li cov vitamin D ib leeg ua ib txoj hauv kev kho mob pob txha.
Therapeutic txoj cai ntawmipriflavonemodulates qhov sib txawv thiab biosynthetic zog ntawm tib neeg cov pob txha pob txha stromal osteoprogenitor cells (BMCs) los ntawm kev txhim kho kev qhia ntawm qee yam tseem ceeb matrix proteins thiab txhawb cov txheej txheem mineralization. Hauv kab lis kev cai nas tsis muaj qhov sib txawv ntawm cov osteocytes, kev kho mob nrog Ipriflavone ntawm koob tshuaj 10 μM cuam tshuam rau cov khoom kawg glycation kawg (AGE) - txhim kho pob txha resorption. Tsis tas li ntawd, kev kho Ipriflavone tuaj yeem txhim kho calcium nqus hauv cov hlwb duodenal. Ipriflavone koob tshuaj-dependently inhibits proliferation thiab DNA synthesis nyob rau hauv MDA -231 hlwb thiab blocks ligand-induced phosphorylation ntawm Tyr845 hauv EGFR. Kev kho Ipriflavone ntawm koob tshuaj 50 μM tsis ua rau apoptosis hauv MDA-231 hlwb.
Ipriflavone yog dawb mus rau yellowish dawb siv lead ua los yog crystalline hmoov, tsw thiab tasteless. Nws yog yooj yim soluble nyob rau hauv chloroform los yog dimethylformamide, kuj soluble nyob rau hauv acetonitrile, acetone los yog ethyl acetate, kuj insoluble nyob rau hauv methanol, anhydrous ethanol los yog anhydrous ether, heev insoluble nyob rau hauv hexane, thiab yuav luag insoluble nyob rau hauv dej. Melting point 115 ~ 117 degree.
Kev ceev faj rau kev siv:
Yog tias koj noj tshuaj tiv thaiv kab mob xws li corticosteroids, methotrexate, thiab lwm yam, nws tsis tsim nyog ntxiv nrog ipriflavone vim nws yuav ua rau lub cev tsis muaj zog.
Yog tias koj tau txais kev kho tshuaj estrogen, koj yuav tsum tsis txhob noj cov tshuaj ipriflavone vim nws ua rau muaj kev pheej hmoo ntawm mob qog noj ntshav mis lossis uterine.
Kev noj tshuaj ntxiv tsis pom zoo rau cov neeg mob peptic ulcers.
Cov neeg muaj kab mob tiv thaiv kab mob xws li AIDS yuav tsum tsis txhob noj.
Cov neeg mob uas muaj cov qe ntshav dawb tsawg yuav tsum tsis txhob noj, lossis raws li kws kho mob qhia.
Eproflavonoids yog metabolized nyob rau hauv ob lub raum, yog li ntawd, cov neeg mob uas mob raum kab mob tsis pom zoo kom coj nws los yog ua raws li tus kws kho mob cov lus qhia.
Yolanda
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